Difference between revisions of "Paroxysmal supraventricular tachycardia pathophysiology"

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===Pathogenesis===
 
===Pathogenesis===
*The exact pathogenesis of [disease name] is not completely understood.
+
'''PSVTs are belived to be caused by abnormalities in impulse formation and conduction pathways.'''
OR
+
 
*It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
+
* '''Impulse Abnormalities:'''
*[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
+
 
*Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
+
The impulse is affected by the previous action potential. In PSVT the abnormal impulse is due to:
*[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
+
 
*The progression to [disease name] usually involves the [molecular pathway].
+
·        Early after-depolarization, which is promoted by:
*The pathophysiology of [disease/malignancy] depends on the histological subtype.
+
 
 +
·        Slow heart rate
 +
 
 +
·        Decreased or increased outward current
 +
 
 +
·        Delayed after-depolarization
 +
 
 +
·        Intracellular Ca++ overload
 +
 
 +
* '''Conduction pathways:'''
 +
 
 +
Different reentry circuits in the heart.
  
 
==Genetics==
 
==Genetics==
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[[Category:Cardiology]]
 
[[Category:Cardiology]]
 
[[Category:Cardiovascular diseases]]
 
[[Category:Cardiovascular diseases]]
[[Category:Arrythmia]]
+
[[Category:Arrhythmia]]
 
[[Category:Emergency medicine]]
 
[[Category:Emergency medicine]]
 
[[Category:Disease]]
 
[[Category:Disease]]

Latest revision as of 06:14, 11 July 2020

Paroxysmal supraventricular tachycardia Microchapters

Home

Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Paroxysmal supraventricular tachycardia from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Study of Choice

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

X-ray

Echocardiography and Ultrasound

CT scan

MRI

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Interventions

Surgery

Primary Prevention

Secondary Prevention

Cost-Effectiveness of Therapy

Future or Investigational Therapies

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Case #1

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Noha Elzeiny, M.B.B.Ch, M.Sc.[2]

Overview

The exact pathogenesis of [disease name] is not fully understood.

OR

It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].

OR

[Pathogen name] is usually transmitted via the [transmission route] route to the human host.

OR

Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.

OR


[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].

OR

The progression to [disease name] usually involves the [molecular pathway].

OR

The pathophysiology of [disease/malignancy] depends on the histological subtype.

Pathophysiology

Physiology

The normal physiology of [name of process] can be understood as follows:

Pathogenesis

PSVTs are belived to be caused by abnormalities in impulse formation and conduction pathways.

  • Impulse Abnormalities:

The impulse is affected by the previous action potential. In PSVT the abnormal impulse is due to:

·        Early after-depolarization, which is promoted by:

·        Slow heart rate

·        Decreased or increased outward current

·        Delayed after-depolarization

·        Intracellular Ca++ overload

  • Conduction pathways:

Different reentry circuits in the heart.

Genetics

[Disease name] is transmitted in [mode of genetic transmission] pattern.

OR

Genes involved in the pathogenesis of [disease name] include:

  • [Gene1]
  • [Gene2]
  • [Gene3]

OR

The development of [disease name] is the result of multiple genetic mutations such as:

  • [Mutation 1]
  • [Mutation 2]
  • [Mutation 3]

Associated Conditions

Conditions associated with [disease name] include:

  • [Condition 1]
  • [Condition 2]
  • [Condition 3]

Gross Pathology

On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

Microscopic Pathology

On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

References




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